Tau is not necessary for amyloid-β–induced synaptic and memory impairments
Author:
Funder
NIH
Canadian Institutes of Health Research grant
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/137040/files/pdf
Reference87 articles.
1. Reducing Endogenous Tau Ameliorates Amyloid ß-Induced Deficits in an Alzheimer's Disease Mouse Model
2. Vossel KA et al Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β J Cell Biol 2015 209 3 419 433
3. Tau Reduction Prevents Aβ-Induced Defects in Axonal Transport
4. Phosphorylation of tau at Y18, but not tau-fyn binding, is required for tau to modulate NMDA receptor-dependent excitotoxicity in primary neuronal culture
5. Tau Protein Is Required for Amyloid -Induced Impairment of Hippocampal Long-Term Potentiation
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