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Inhibition of airway remodeling in IL-5–deficient mice

Published:2004-02-15 Issue:4 Volume:113 Page:551-560
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Cho Jae Youn,Miller Marina,Baek Kwang Je,Han Ji Won,Nayar Jyothi,Lee Sook Young,McElwain Kirsti,McElwain Shauna,Friedman Stephanie,Broide David H.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference34 articles.

1. Eosinophilia

2. Fast Flowing Eosinophils

3. Recombinant human interleukin 5 is a selective activator of human eosinophil function.

4. Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model.

5. Interleukin-5 Expression in the Lung Epithelium of Transgenic Mice Leads to Pulmonary Changes Pathognomonic of Asthma

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1. Navigating the Terrain: Type 2 Cytokines and Biologic intervention in Severe Eosinophilic Asthma;Eosinophils and Their Role in Human Health and Disease [Working Title];2024-08-23

2. Role of IL‐5 in asthma and airway remodelling;Clinical & Experimental Allergy;2024-06-27

3. Airway epithelial overexpressed cathepsin K induces airway remodelling through epithelial–mesenchymal trophic unit activation in asthma;British Journal of Pharmacology;2024-06-09

4. IL-4 regulates neutrophilic pulmonary inflammation in a mouse model of bronchial asthma;Cytokine;2024-06

5. Adverse events of anti-IL-5 drugs in patients with eosinophilic asthma: a meta-analysis of randomized controlled trials and real-world evidence-based assessments;BMC Pulmonary Medicine;2024-02-03

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