A transgenic mouse model for HLA-B*57:01–linked abacavir drug tolerance and reactivity
Author:
Funder
FDA and NIH
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/99321/files/pdf
Reference70 articles.
1. T Cell–Mediated Hypersensitivity Reactions to Drugs
2. Genetic variations in HLA-B region and hypersensitivity reactions to abacavir
3. Association between presence of HLA-B*5701, HLA-DR7 , and HLA-DQ3 and hypersensitivity to HIV-1 reverse-transcriptase inhibitor abacavir
4. HLA-B*5701 genotype is a major determinant of drug-induced liver injury due to flucloxacillin
5. HLA-B*57:01 Confers Susceptibility to Pazopanib-Associated Liver Injury in Patients with Cancer
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3. Development of mouse models with restricted HLA-B∗57:01 presentation for the study of flucloxacillin-driven T-cell activation and tolerance in liver injury;Journal of Allergy and Clinical Immunology;2023-08
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