Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia-Reference-Cited by-同舟云学术

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Published:2003-03-01 Issue:5 Volume:111 Page:649-658
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Maynard Sharon E.,Min Jiang-Yong,Merchan Jaime,Lim Kee-Hak,Li Jianyi,Mondal Susanta,Libermann Towia A.,Morgan James P.,Sellke Frank W.,Stillman Isaac E.,Epstein Franklin H.,Sukhatme Vikas P.,Karumanchi S. Ananth

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference47 articles.

1. Preeclampsia: What we know and what we do not know

2. Pathogenesis and genetics of pre-eclampsia

3. Excessive placental secretion of neurokinin B during the third trimester causes pre-eclampsia

4. Identification of a Natural Soluble Form of the Vascular Endothelial Growth Factor Receptor, FLT-1, and Its Heterodimerization with KDR

5. Structure and Function of VEGF/VEGF-receptor System Involved in Angiogenesis.

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