Focal expression of interleukin-2 does not break unresponsiveness to "self" (viral) antigen expressed in beta cells but enhances development of autoimmune disease (diabetes) after initiation of an anti-self immune response.
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Cited by 53 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Autoimmunity in picornavirus infections;Current Opinion in Virology;2016-02
2. Mechanisms of Beta Cell Dysfunction Associated With Viral Infection;Current Diabetes Reports;2015-08-18
3. Mucosal Immunity;Mandell, Douglas, and Bennett's Principles and Practice of Infectious Diseases;2015
4. Contributions of Viruses and Immunity as Causes of Diabetes and Development of Strategies for Treatment and Prevention of Autoimmune Disease;Molecular Mimicry, Microbes, and Autoimmunity;2014-04-08
5. Immunology in the clinic review series: focus on type 1 diabetes and viruses: the role of viruses in type 1 diabetes: a difficult dilemma;Clinical and Experimental Immunology;2012-03-02
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