Loss of acinar cell IKKα triggers spontaneous pancreatitis in mice-Reference-Cited by-同舟云学术

Loss of acinar cell IKKα triggers spontaneous pancreatitis in mice

Published:2013-04-08 Issue:5 Volume:123 Page:2231-2243
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Li Ning,Wu Xuefeng,Holzer Ryan G.,Lee Jun-Hee,Todoric Jelena,Park Eek-Joong,Ogata Hisanobu,Gukovskaya Anna S.,Gukovsky Ilya,Pizzo Donald P.,VandenBerg Scott,Tarin David,Atay Çiǧdem,Arkan Melek C.,Deerinck Thomas J.,Moscat Jorge,Diaz-Meco Maria,Dawson David,Erkan Mert,Kleeff Jörg,Karin Michael

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference62 articles.

1. Chronic Pancreatitis: Genetics and Pathogenesis

2. Molecular biology of pancreatic cancer

3. Impaired autophagic flux mediates acinar cell vacuole formation and trypsinogen activation in rodent models of acute pancreatitis

4. Intra-acinar Trypsinogen Activation Mediates Early Stages of Pancreatic Injury but Not Inflammation in Mice With Acute Pancreatitis

5. Intracellular activation of trypsinogen in transgenic mice induces acute but not chronic pancreatitis

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