Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease-Reference-Cited by-同舟云学术

Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney disease

Published:2007-08-01 Issue:8 Volume:117 Page:2095-2104
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Sever Sanja,Altintas Mehmet M.,Nankoe Sharif R.,Möller Clemens C.,Ko David,Wei Changli,Henderson Joel,del Re Elizabetta C.,Hsing Lianne,Erickson Ann,Cohen Clemens D.,Kretzler Matthias,Kerjaschki Dontscho,Rudensky Alexander,Nikolic Boris,Reiser Jochen

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference39 articles.

1. Why is proteinuria an ominous biomarker of progressive kidney disease?

2. Hereditary Proteinuria Syndromes and Mechanisms of Proteinuria

3. Podocyte Migration during Nephrotic Syndrome Requires a Coordinated Interplay between Cathepsin L and α3 Integrin

4. The cysteine proteinase inhibitor, E-64, reduces proteinuria in an experimental model of glomerulonephritis

5. A Cathepsin L Isoform that Is Devoid of a Signal Peptide Localizes to the Nucleus in S Phase and Processes the CDP/Cux Transcription Factor

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