Impaired lymphocyte development and antibody class switching and increased malignancy in a murine model of DNA ligase IV syndrome-Reference-Cited by-同舟云学术

Impaired lymphocyte development and antibody class switching and increased malignancy in a murine model of DNA ligase IV syndrome

Published:2009-06-01 Issue:6 Volume:119 Page:1696-1705
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Nijnik Anastasia,Dawson Sara,Crockford Tanya L.,Woodbine Lisa,Visetnoi Supawan,Bennett Sophia,Jones Margaret,Turner Gareth D.,Jeggo Penelope A.,Goodnow Christopher C.,Cornall Richard J.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference58 articles.

1. Mechanism and regulation of human non-homologous DNA end-joining

2. Cernunnos, a Novel Nonhomologous End-Joining Factor, Is Mutated in Human Immunodeficiency with Microcephaly

3. XLF Interacts with the XRCC4-DNA Ligase IV Complex to Promote DNA Nonhomologous End-Joining

4. DNA Ligase IV Mutations Identified in Patients Exhibiting Developmental Delay and Immunodeficiency

5. A patient with mutations in DNA Ligase IV: Clinical features and overlap with Nijmegen breakage syndrome

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