Four individually druggable MET hotspots mediate HGF-driven tumor progression-Reference-Cited by-同舟云学术

Four individually druggable MET hotspots mediate HGF-driven tumor progression

Published:2014-05-27 Issue:7 Volume:124 Page:3172-3186
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Basilico Cristina,Hultberg Anna,Blanchetot Christophe,de Jonge Natalie,Festjens Els,Hanssens Valérie,Osepa Sjudry-Ilona,De Boeck Gitte,Mira Alessia,Cazzanti Manuela,Morello Virginia,Dreier Torsten,Saunders Michael,de Haard Hans,Michieli Paolo

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference52 articles.

1. Invasive growth: a MET-driven genetic programme for cancer and stem cells

2. Met, metastasis, motility and more

3. Drug development of MET inhibitors: targeting oncogene addiction and expedience

4. Hypoxia promotes invasive growth by transcriptional activation of the met protooncogene

5. The HGF-Met Signaling Axis: Emerging Themes and Targets of Inhibition

Cited by 43 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Phage display for discovery of anticancer antibodies;New Biotechnology;2024-11

2. MET Oncogene Targeting for Cancer Immunotherapy;International Journal of Molecular Sciences;2024-06-01

3. Hepatocellular Carcinoma: Old and Emerging Therapeutic Targets;Cancers;2024-02-23

4. The molecular structure and function of fibrocystin, the key gene product implicated in autosomal recessive polycystic kidney disease (ARPKD);Annals of Human Genetics;2023-10-31

5. HGF/c-MET: A Potential Target for the Treatment of Various Cancers;Current Enzyme Inhibition;2023-08-08