CYP24 inhibition as a therapeutic target in FGF23-mediated renal phosphate wasting disorders-Reference-Cited by-同舟云学术

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CYP24 inhibition as a therapeutic target in FGF23-mediated renal phosphate wasting disorders

Published:2016-01-19 Issue:2 Volume:126 Page:667-680
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Bai Xiuying,Miao Dengshun,Xiao Sophia,Qiu Dinghong,St-Arnaud René,Petkovich Martin,Gupta Ajay,Goltzman David,Karaplis Andrew C.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference66 articles.

1. FGF23 and Syndromes of Abnormal Renal Phosphate Handling

2. Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

3. Role of FGF23 in vitamin D and phosphate metabolism: Implications in chronic kidney disease

4. Human Fibroblast Growth Factor-23 Mutants Suppress Na+-dependent Phosphate Co-transport Activity and 1α,25-Dihydroxyvitamin D3 Production

5. The Autosomal Dominant Hypophosphatemic Rickets R176Q Mutation in Fibroblast Growth Factor 23 Resists Proteolytic Cleavage and Enhances in Vivo Biological Potency

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1. X-linked hypophosphataemia;The Lancet;2024-08

2. Genetic causes of hypophosphatemia;Minerva Medica;2024-06

3. Emerging Role of Gallium-68 DOTANOC PET/CT Guided Radiofrequency Ablation in the Treatment of Tumor-induced Osteomalacia;JCEM Case Reports;2024-04-23

4. Intestinal-Specific Cyp24a1 Deletion Induces Toll-Like Receptor-4 Expression and Reduces Chemotherapy-Induced Intestinal Injury in Mice;2024

5. Vitamin D and FGF23;Feldman and Pike' s Vitamin D;2024

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