FcγRIIB regulates autoantibody responses by limiting marginal zone B cell activation
Author:
Funder
National Institutes of Health
American Autoimmune Related Disease Association
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Link
https://www.jci.org/articles/view/157250/files/pdf
Reference50 articles.
1. FcγRIIB in autoimmunity and infection: evolutionary and therapeutic implications
2. Partially Distinct Molecular Mechanisms Mediate Inhibitory FcγRIIB Signaling in Resting and Activated B Cells
3. Autoimmune-prone mice share a promoter haplotype associated with reduced expression and function of the Fc receptor FcγRII
4. Decreased transcription of the human FCGR2B gene mediated by the -343 G/C promoter polymorphism and association with systemic lupus erythematosus
5. The Role of Activating Protein 1 in the Transcriptional Regulation of the Human FCGR2B Promoter Mediated by the -343 G → C Polymorphism Associated with Systemic Lupus Erythematosus
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