KIS protects against adverse vascular remodeling by opposing stathmin-mediated VSMC migration in mice
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Reference31 articles.
1. Bone marrow–derived immune cells regulate vascular disease through a p27Kip1-dependent mechanism
2. Stathmin interaction with a putative kinase and coiled-coil-forming protein domains.
3. A growth factor-dependent nuclear kinase phosphorylates p27Kip1 and regulates cell cycle progression
4. KIS Is a Protein Kinase with an RNA Recognition Motif
5. Specific Ser-Pro phosphorylation by the RNA-recognition motif containing kinase KIS
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