Myofibrillar protein turnover: The proteasome and the calpains1,2
Author:
Publisher
Oxford University Press (OUP)
Subject
Genetics,Animal Science and Zoology,General Medicine,Food Science
Link
http://academic.oup.com/jas/article-pdf/86/suppl_14/E19/23673450/e19.pdf
Reference74 articles.
1. Calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubes;Alderton;J. Biol. Chem.,2000
2. Delay of muscle degeneration and necrosis in mdx mice by calpain inhibition;Badalamente;Muscle Nerve,2000
3. Effect of β-agonists on expression of calpain and calpastatin activity in skeletal muscle;Bardsley;Biochemie,1992
4. Diabetes enhances calpain degradation of cardiac myofibrils and easily releasable myofilaments;Belcastro,1991
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