Elevated Factor XI Activity Levels Are Associated With an Increased Odds Ratio for Cerebrovascular Events
Author:
Publisher
Oxford University Press (OUP)
Subject
General Medicine
Link
http://academic.oup.com/ajcp/article-pdf/126/3/411/4987900/ajcpath126-0411.pdf
Reference20 articles.
1. Gailani D, Broze GJ Jr. Factor XI activation in a revised model of blood coagulation. Science. 1991;253:909-912.
2. Walsh PN. Platelets and factor XI bypass the contact system of blood coagulation. Thromb Haemost. 1999;82:234-242.
3. Baglia FA, Walsh PN. Thrombin-mediated feedback activation of factor XI on the activated platelet surface is preferred over contact activation by factor XIIa or XIa. J Biol Chem. 2000;275:20514-20519.
4. Von dem Borne PA, Meijers JCM, Bouma BN. Feedback activation of factor XI by thrombin in plasma results in additional formation of thrombin that protects fibrin clots from fibrinolysis. Blood. 1995;86:3035-3042.
5. Von dem Borne PA, Bajzar L, Meijers JCM, et al. Thrombin-mediated activation of factor XI results in a thrombin-activatable fibrinolysis inhibitor-dependent inhibition of fibrinolysis. J Clin Invest. 1997;99:2323-2327.
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