FLT3andNPM1Mutations in Myelodysplastic Syndromes
Author:
Publisher
Oxford University Press (OUP)
Subject
General Medicine
Link
http://academic.oup.com/ajcp/article-pdf/135/1/62/5003301/ajcpath135-0062.pdf
Reference23 articles.
1. Olney HJ Le Beau MM . Evaluation of recurring cytogenetic abnormalities in the treatment of myelodysplastic syndromes. Leuk Res. 2007;4:427–434.
2. Drexler HG . Expression of FLT3 receptor and response to FLT3 ligand by leukemic cells. Leukemia. 1996;4:588–599.
3. Kottaridis PD Gale RE Frew ME The presence of a FLT3 internal tandem duplication in patients with acute myeloid leukemia (AML) adds important prognostic information to cytogenetic risk group and response to the first cycle of chemotherapy: analysis of 854 patients from the United Kingdom Medical Research Council AML 10 and 12 trials. Blood. 2001;6:1752–1759.
4. Bacher U Haferlach C Kern W Prognostic relevance of FLT3-TKD mutations in AML: the combination matters: an analysis of 3082 patients. Blood. 2008;5:2527–2537.
5. Schneider F Hoster E Unterhalt M NPM1 but not FLT3-ITD mutations predict early blast cell clearance and CR rate in patients with normal karyotype AML (NK-AML) or high-risk myelodysplastic syndrome (MDS). Blood. 2009;21:5250–5253.
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