Arachidonic acid metabolites-induced activation of group 2 innate lymphoid cell in eosinophilic upper airway inflammation
Author:
Affiliation:
1. Department of Otorhinolaryngology, Shiga University of Medical Science
Publisher
Japan Society of Immunology and Allergology in Otolaryngology
Link
https://www.jstage.jst.go.jp/article/jjiao/37/3/37_217/_pdf
Reference38 articles.
1. 1) Spits H, Artis D, et al. Innate lymphoid cells—a proposal for uniform nomenclature. Nat Rev Immunol. 2013; 13: 145–9.
2. 2) Bal SM, Bernink JH, et al. IL-1β, IL-4 and IL-12 control the fate of group 2 innate lymphoid cells in human airway inflammation in the lungs. Nat Immunol. 2016; 17: 636–45.
3. 3) Zhang K, Xu X, et al. Cutting Edge: Notch Signaling Promotes the Plasticity of Group-2 Innate Lymphoid Cells. J Immunol. 2017; 198: 1798–803.
4. 4) Cai T, Qui J, et al. IL-17-producing ST2+ group 2 innate lymphoid cells play a pathogenic role in lung inflammation. J Allergy Clin Immunol. 2019; 143: 229–44.
5. 5) Xue L, Salimi M, et al. Prostaglandin D2 activates group 2 innate lymphoid cells through chemoattractant receptor-homologous molecule expressed on TH2 cells. J Allergy Clin Immunol. 2014; 133: 1184–94.
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