The Relationship between Prohibitin 1 Expression, Hepatotoxicity Induced by Acetaminophen, and Hepatoprotection by S-Adenosylmethionine in AML12 Cells-Reference-Cited by-同舟云学术

The Relationship between Prohibitin 1 Expression, Hepatotoxicity Induced by Acetaminophen, and Hepatoprotection by S-Adenosylmethionine in AML12 Cells

Published:2022-09-26 Issue:11 Volume:32 Page:1447-1453
ISSN:1017-7825
Container-title:Journal of Microbiology and Biotechnology
language:en
Short-container-title:J. Microbiol. Biotechnol.

Author:

Cho Eunhye¹,Jung Soohan²,Kim Jina³,Ko Kwang Suk¹⁴

Affiliation:

1. Department of Nutritional Science and Food Management, Ewha Womans University, Seoul 03760, Republic of Korea

2. Department of Integrated Biomedical and Life Science, Korea University, Seoul 02841, Republic of Korea

3. Department of Human Biology, University of Southern California, Los Angeles, CA 90089, USA

4. Graduate Program in System Health Science and Engineering, Ewha Womans University, Seoul 03760, Republic of Korea

Funder

National Research Foundation of Korea

Ministry of Science and ICT

BK21 FOUR

Ministry of Education

Publisher

Korean Society for Microbiology and Biotechnology

Subject

Applied Microbiology and Biotechnology,General Medicine,Biotechnology

Link

http://pdf.medrang.co.kr/JMB2/2022/032/jmb-32-11-1447.pdf

Reference28 articles.

1. Metabolism and Disposition of Acetaminophen: Recent Advances in Relation to Hepatotoxicity and Diagnosis

2. Betaine protects mice against acetaminophen hepatotoxicity possibly via mitochondrial complex II and glutathione availability

3. Detoxifying effect of pyridoxine on acetaminophen-induced hepatotoxicity via suppressing oxidative stress injury

4. Acetaminophen toxicity results in site-specific mitochondrial damage in isolated mouse hepatocytes.

5. Acetaminophen-Induced Oxidant Stress and Cell Injury in Cultured Mouse Hepatocytes: Protection by N-Acetyl Cysteine

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