Role of Fas–Fas Ligand Interactions in 2,3,7,8-Tetrachlorodibenzo- p-dioxin (TCDD)-Induced Immunotoxicity: Increased Resistance of Thymocytes from Fas-Deficient (lpr) and Fas Ligand-Defective (gld) Mice to TCDD-Induced Toxicity

Author:

Kamath Arati B.,Camacho Iris,Nagarkatti Prakash S.,Nagarkatti Mitzi

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference33 articles.

1. Effect of in vivo administered 2,3,7,8-tetrachlorodibenzo-p-dioxin on DNA-binding activities of nuclear transcription factors in liver of guinea pigs;Ashida;J. Biochem. Mol. Toxicol.,1998

2. Exposure to tetrachlorodibenzo-p-dioxin (TCDD) alters fetal thymocyte maturation;Blaylock;Toxicol. Appl. Pharmacol.,1992

3. Dioxin induces transforming growth factor-alpha in human keratinocytes;Choi;J. Biol. Chem.,1991

4. T cell deletion in high antigen dose therapy of autoimmune encephalomyelitis;Critchfield;Science,1994

5. Modulation of growth factor expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin;Dohr;Exp. Clin. Immunogenet.,1994

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