Chronic Hyperammonemia in Rats Impairs Activation of Soluble Guanylate Cyclase in Neurons and in Lymphocytes: A Putative Peripheral Marker for Neurological Alterations-Reference-Cited by-同舟云学术

Chronic Hyperammonemia in Rats Impairs Activation of Soluble Guanylate Cyclase in Neurons and in Lymphocytes: A Putative Peripheral Marker for Neurological Alterations

Published:1999-04 Issue:2 Volume:257 Page:405-409
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Miñana Marı́a-Dolores,Corbalán Regina,Montoliu Carmina,Teng Che-Ming,Felipo Vicente

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. The Release and Neosynthesis of Glutamic Acid Are Increased in Experimental Models of Hepatic Encephalopathy

2. Glutamatergic synaptic dysfunction in hyperammonemic syndromes

3. Ammonium acetate inhibits ionotropic receptors and differentially affects metabotropic receptors for glutamate

4. Hepatic Encephalopathy in Acute Liver Failure: Role of the Glutamate System

5. Glutamate and Muscarinic Receptors in the Molecular Mechanisms of Acute Ammonia Toxicity and of Its Prevention

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4. Lymphocyte cytochrome c oxidase, cyclic GMP and cholinergic muscarinic receptors as peripheral indicators of carbon monoxide neurotoxicity after acute and repeated exposure in the rat;Life Sciences;2006-03

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