Analysis of p16INK4aand Its Interaction with CDK4
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Cited by 32 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Comparison of mutation landscapes of pretreatment versus recurrent squamous cell carcinoma of the oral cavity: The possible mechanism of resistance to standard treatment;Cancer Reports;2024-03
2. Comprehensive Analysis Revealed that CDKN2A is a Biomarker for Immune Infiltrates in Multiple Cancers;Frontiers in Cell and Developmental Biology;2021-12-23
3. C-terminal domain of p16INK4a is adequate in inducing cell cycle arrest, growth inhibition and CDK4/6 interaction similar to the full length protein in HT-1080 fibrosarcoma cells;Journal of Cellular Biochemistry;2010-11-29
4. ATP‐Noncompetitive Inhibitors of CDK–Cyclin Complexes;ChemMedChem;2009-01-05
5. Genetic and Epigenetic Changes in Mammary Epithelial Cells Identify a Subpopulation of Cells Involved in Early Carcinogenesis;Cold Spring Harbor Symposia on Quantitative Biology;2005-01-01
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