Ouabain Suppresses ATP Elevation in Response to Fuel Secretagogues in Pancreatic Islets
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference23 articles.
1. The pancreatic beta-cell as a fuel sensor: an electrophysiologist's viewpoint
2. New insights into pancreatic β-cell metabolic signaling in insulin secretion
3. Glucose dependent K+-channels in pancreatic?-cells are regulated by intracellular ATP
4. A metabolite-regulated potassium channel in rat pancreatic B cells.
5. Modulation of dihydropyridine-sensitive Ca2+ channels by glucose metabolism in mouse pancreatic β -cells
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Role of endogenous ROS production in impaired metabolism-secretion coupling of diabetic pancreatic β cells;Progress in Biophysics and Molecular Biology;2011-11
2. Molecular mechanism of impaired metabolism-secretion coupling in diabetic pancreatic β cells;Diabetology International;2011-09-30
3. Impaired metabolism–secretion coupling in pancreatic β-cells: Role of determinants of mitochondrial ATP production;Diabetes Research and Clinical Practice;2007-09
4. Ouabain Suppresses Glucose-Induced Mitochondrial ATP Production and Insulin Release by Generating Reactive Oxygen Species in Pancreatic Islets;Diabetes;2002-08-01
5. Modulation of islet ATP content by inhibition or stimulation of the Na+/K+ pump;European Journal of Pharmacology;2001-08
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