The Ang II-Induced Growth of Vascular Smooth Muscle Cells Involves a Phospholipase D-Mediated Signaling Mechanism
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Biochemistry,Biophysics
Reference38 articles.
1. Sustained diacylglycerol formation from inositol phospholipids in angiotensin II-stimulated vascular smooth muscle cells.
2. Angiotensin II stimulation of rapid protein tyrosine phosphorylation and protein kinase activation in rat aortic smooth muscle cells.
3. The role of protein kinase C in activation and termination of mitogen-activated protein kinase activity in angiotensin II-stimulated rat aortic smooth-muscle cells
4. ANG II-induced translocation of cytosolic PLA2 to the nucleus in vascular smooth muscle cells
5. Stimulated Mitogen-activated Protein Kinase Is Necessary but Not Sufficient for the Mitogenic Response to Angiotensin II
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1. Angiotensin II-dependent growth of vascular smooth muscle cells requires transactivation of the epidermal growth factor receptor via a cytosolic phospholipase A2-mediated release of arachidonic acid;Archives of Biochemistry and Biophysics;2010-06
2. Roles of brain phosphatidylinositol-specific phospholipase C and diacylglycerol lipase in centrally administered histamine-induced adrenomedullary outflow in rats;European Journal of Pharmacology;2007-10
3. ANG II stimulates phospholipase D through PKCζ activation in VSMC: implications in adhesion, spreading, and hypertrophy;American Journal of Physiology-Heart and Circulatory Physiology;2006-01
4. ANG II-induced neointimal growth is mediated via cPLA2- and PLD2-activated Akt in balloon-injured rat carotid artery;American Journal of Physiology-Heart and Circulatory Physiology;2005-12
5. Modification of intracellular free calcium in cultured A10 vascular smooth muscle cells by exogenous phosphatidic acid;Biochemical Pharmacology;2003-06
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