Inhibitory Effects of Streptozotocin, Tumor Necrosis Factor-α, and Interleukin-1β on Glucokinase Activity in Pancreatic Islets and Gene Expression of GLUT2 and Glucokinase-Reference-Cited by-同舟云学术

Inhibitory Effects of Streptozotocin, Tumor Necrosis Factor-α, and Interleukin-1β on Glucokinase Activity in Pancreatic Islets and Gene Expression of GLUT2 and Glucokinase

Published:1999-02 Issue:2 Volume:362 Page:217-224
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Park Chun,Kim Jeong-Ran,Shim Jae-Kyoung,Kang Bong-Seok,Park Young-Guk,Nam Kyung-Soo,Lee Young-Choon,Kim Cheorl-Ho

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference48 articles.

1. Functional characteristics of cultured mouse pancreatic islets following exposure to different streptozotocin concentrations

2. Preferential Reduction of Insulin Production in Mouse Pancreatic Islets Maintained in Culture after Streptozotocin Exposure*

3. Persistent impairment of the insulin response to glucose both in vivo and in vitro after streptozotocin exposure: Studies with grafted pancreatic islets and islets maintained in culture

4. Survival and B-Cell Function of Mouse Pancreatic Islets Maintained in Culture after Concomitant Exposure to Streptozotocin and Nicotinamide

5. Interleukin-1β inhibits glucokinase activity in clonal HIT-T15 β-cells

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