Effect of Transforming Growth Factor-β1 on the Cell Growth and Epstein–Barr Virus Reactivation in EBV-Infected Epithelial Cell Lines

Author:

Fukuda Makoto,Ikuta Kazufumi,Yanagihara Kazuyoshi,Tajima Masako,Kuratsune Hirohiko,Kurata Takeshi,Sairenji Takeshi

Publisher

Elsevier BV

Subject

Virology

Reference42 articles.

1. Latent membrane protein-1 induces cyclin D2 expression, pRb hyperphosphorylation, and loss of TGF-beta 1-mediated growth inhibition in EBV-positive B cells;Arvanitakis;J. Immunol.,1995

2. Physicochemical activation of recombinant latent transforming growth factor-beta's 1, 2, and 3;Brown;Growth Factors,1990

3. Polymorphic proteins encoded within BZLF1 of defective and standard Epstein–Barr viruses disrupt latency;Countryman;J. Virol.,1987

4. Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene;Dennler;EMBO J.,1998

5. Endogenous TGF-beta contributes to the induction of the EBV lytic cycle in two Burkitt lymphoma cell lines;di Renzo;Int. J. Cancer,1994

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