The Role of the J domain of SV40 Large T in Cellular Transformation
Author:
Publisher
Elsevier BV
Subject
Pharmacology,General Immunology and Microbiology,Applied Microbiology and Biotechnology,General Medicine,Bioengineering,Biotechnology
Reference36 articles.
1. Mutants with changes within or near a hydrophobic region of simian virus 40 large tumor antigen are defective for binding cellular protein p53;Peden;Virology,1989
2. The large tumor antigen of simian virus 40 encodes at least two distinct transforming functions;Srinivasan;J Virol,1989
3. Transformation of a continuous rat embryo fibroblast cell line requires three separate domains of simian virus 40 large T antigen;Zhu;J Virol,1992
4. Association of p53 binding and immortalization of primary C57BL/6 mouse embryo fibroblasts by using simian virus 40 T-antigen mutants bearing internal overlapping deletion mutations;Kierstead;J Virol,1993
5. In vitro mutagenesis of a putative DNA binding domain of SV40 large-T;Kalderon;Virology,1984
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