Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression in Mice Causes Mitochondrial Vacuolization, Axonal Degeneration, and Premature Motoneuron Death and Accelerates Motoneuron Disease in Mice Expressing a Familial Amyotrophic Lateral Sclerosis Mutant SOD1-Reference-Cited by-同舟云学术

Human Cu/Zn Superoxide Dismutase (SOD1) Overexpression in Mice Causes Mitochondrial Vacuolization, Axonal Degeneration, and Premature Motoneuron Death and Accelerates Motoneuron Disease in Mice Expressing a Familial Amyotrophic Lateral Sclerosis Mutant SOD1

Published:2000-12 Issue:6 Volume:7 Page:623-643
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Jaarsma Dick,Haasdijk Elize D.,Grashorn J.A.C.,Hawkins Richard,van Duijn Wim,Verspaget Hein W.,London Jacqueline,Holstege Jan C.

Publisher

Elsevier BV

Subject

Neurology

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