The Role of Inhibition of Gap Junctional Intercellular Communication in Rodent Liver Tumor Induction by Phthalates: Review of Data on Selected Phthalates and the Potential Relevance to Man

Author:

McKee Richard H.

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference46 articles.

1. Reversibility of phenobarbital and DEHP-induced inhibition of gap junction intercellular communication (GJIC) and induction of DNA synthesis in the rat;Ackley;Toxicologist,1999

2. Mechanistically based human hazard assessment of peroxisome proliferator-induced hepatocarcinogenesis;Ashby;Hum. Exp. Toxicol.,1994

3. Gap junctional intercellular communication (GJIC) studies on 5 phthalate monoesters in hepatocytes of four species: Implications for cancer risk assessment;Baker;Toxicologist,1996

4. Results in the L5178Y mouse lymphoma and the in vitro transformation of Balb/3T3 cell assays for eight phthalate esters;Barber;J. Appl. Toxicol.,2000

5. Species differences in the response of cultured hepatocytes to phthalate esters;Benford;Food Chem. Toxicol.,1986

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