The Long Terminal Repeats of Human Immunodeficiency Virus Type-1 and Human T-cell Leukemia Virus Type-I Are Activated by 12-O-Tetradecanoylphorbol- 13-acetate through Different Pathways-Reference-Cited by-同舟云学术

The Long Terminal Repeats of Human Immunodeficiency Virus Type-1 and Human T-cell Leukemia Virus Type-I Are Activated by 12-O-Tetradecanoylphorbol- 13-acetate through Different Pathways

Published:1997-06 Issue:2 Volume:232 Page:337-344
ISSN:0042-6822
Container-title:Virology
language:en
Short-container-title:Virology

Author:

Mor-Vaknin Nirit,Torgeman Amram,Galron Dalia,Löchelt Martin,Flügel Rolf M.,Aboud Mordechai

Publisher

Elsevier BV

Subject

Virology

Reference45 articles.

1. Regulatory elements and leukemogenicity of HTLV-I and HTLV-II;Aboud;J. Viral Dis.,1993

2. The jun proto-oncogene is positively autoregulated by its product Jun/AP-1;Angel;Cell,1988

3. Ha-ras augments c-Jun activity and stimulates phosphorylation of its activation domain;Binetruy;Nature (London),1991

4. The product of the c-ets-1 proto-oncogene and the related ets2 act as transcriptional activators of the long terminal repeat of the human T-cell leukemia virus HTLV-1;Bosselut;EMBO. J.,1990

5. Myb protein binds to multiple site in the human T-lymphotropic virus type 1 long terminal repeat and transactivates LTR-mediated expression;Bosselut;Virology,1992

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