Resistance to Malarial Infection Is Achieved by the Cooperation of NK1.1+ and NK1.1− Subsets of Intermediate TCR Cells Which Are Constituents of Innate Immunity-Reference-Cited by-同舟云学术

Resistance to Malarial Infection Is Achieved by the Cooperation of NK1.1+ and NK1.1− Subsets of Intermediate TCR Cells Which Are Constituents of Innate Immunity

Published:2001-08 Issue:2 Volume:211 Page:96-104
ISSN:0008-8749
Container-title:Cellular Immunology
language:en
Short-container-title:Cellular Immunology

Author:

Mannoor M.Kaiissar,Weerasinghe Anura,Halder Ramesh C.,Reza Sufi,Morshed M.,Ariyasinghe Anoja,Watanabe Hisami,Sekikawa Hiroho,Abo Toru

Publisher

Elsevier BV

Subject

Immunology

Reference58 articles.

1. Liver CD4−CD8−NK1.1+TCRαβ intermediate cells increase during experimental malaria infection and are able to exhibit inhibitory activity against the parasite liver stage in vitro;Pied;J. Immunol.,2000

2. Association of intermediate T cell receptor cells, mainly their NK1.1− subset, with protection from malaria;Weerasinghe;Cell. Immunol.,2001

3. Cutting edge. The IgG response to the circumsporozoite protein is MHC class II-dependent and CD1d-independent: Exploring the role of GPIs in NK T cell activation and antimalarial responses;Molano;J. Immunol.,2000

4. α-Galactosylceramide-activated Vα14 natural killer T cells mediate protection against murine malaria;Gonzalez-Aseguinolaza;Proc. Natl. Acad. Sci. USA,2000

5. The implications for malaria vaccine programs if memory T cells from non-exposed humans can respond to malaria antigens;Good;Curr. Opin. Immunol.,1991

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