Pulmonary Vascular Smooth Muscle Relaxation by cGMP- versus cAMP-Mediated Mechanisms
Author:
Publisher
Elsevier BV
Subject
Surgery
Cited by 26 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Role of the Prostanoid EP4 Receptor in Iloprost-mediated Vasodilatation in Pulmonary Hypertension;American Journal of Respiratory and Critical Care Medicine;2008-07-15
2. IL-1β, BK, and TGF-β1attenuate PGI2-mediated cAMP formation in human pulmonary artery smooth muscle cells by multiple mechanisms involving p38 MAP kinase and PKA;American Journal of Physiology-Lung Cellular and Molecular Physiology;2008-03
3. Interleukin-1β, Transforming Growth Factor-β 1 , and Bradykinin Attenuate Cyclic AMP Production by Human Pulmonary Artery Smooth Muscle Cells in Response to Prostacyclin Analogues and Prostaglandin E 2 by Cyclooxygenase-2 Induction and Downregulation of Adenylyl Cyclase Isoforms 1, 2, and 4;Circulation Research;2004-02-20
4. The effects of selective phosphodiesterase III and V inhibitors on adrenergic and non-adrenergic, non-cholinergic relaxation responses of guinea-pig pulmonary arteries;Autonomic and Autacoid Pharmacology;2003-04
5. Role of the Nitric Oxide-cGMP System in the Regulation of Ductus Arteriosus Patency in Fetal Rats.;Journal of Veterinary Medical Science;1999
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