Evidence That Nucleotide Excision Repair Is Attenuated in Bax-Deficient Mammalian Cells Following Ultraviolet Irradiation-Reference-Cited by-同舟云学术

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Evidence That Nucleotide Excision Repair Is Attenuated in Bax-Deficient Mammalian Cells Following Ultraviolet Irradiation

Published:2002-08-15 Issue:2 Volume:278 Page:158-165
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Cho S

Publisher

Elsevier BV

Subject

Cell Biology

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The secret life of Bcl-2: Apoptosis-independent inhibition of DNA repair by Bcl-2 family members;Mutation Research/Reviews in Mutation Research;2012-10

2. Cooperation of Ha-ras and Bcl-2 during multistep skin carcinogenesis;Molecular Carcinogenesis;2007

3. Molecular mediators of cell death in multistep carcinogenesis: a path to targeted therapy;Cell Death & Differentiation;2006-06-09

4. Androgen and Its Receptor Promote Bax-Mediated Apoptosis;Molecular and Cellular Biology;2006-03

5. Bax and Bid, two proapoptotic Bcl-2 family members, inhibit homologous recombination, independently of apoptosis regulation;Oncogene;2006-01-16

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