Differential Effects of Nonselective Nitric Oxide Synthase (NOS) and Selective Inducible NOS Inhibition on Hepatic Necrosis, Apoptosis, ICAM-1 Expression, and Neutrophil Accumulation during Endotoxemia-Reference-Cited by-同舟云学术

Differential Effects of Nonselective Nitric Oxide Synthase (NOS) and Selective Inducible NOS Inhibition on Hepatic Necrosis, Apoptosis, ICAM-1 Expression, and Neutrophil Accumulation during Endotoxemia

Published:1997-10 Issue:5 Volume:1 Page:404-416
ISSN:1089-8603
Container-title:Nitric Oxide
language:en
Short-container-title:Nitric Oxide

Author:

Ou Junhai,Carlos Timothy M.,Watkins Simon C.,Saavedra Joseph E.,Keefer Larry K.,Kim Young-Myeong,Harbrecht Brian G.,Billiar Timothy R.

Publisher

Elsevier BV

Subject

Cancer Research,Clinical Biochemistry,Physiology,Biochemistry

Reference60 articles.

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5. Activation of the 55 kDa TNF receptor is necessary and sufficient for TNF-induced liver failure, hepatocyte apoptosis, and nitrite release;Leist;J. Immunol.,1995

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