Cycling Werner's Syndrome Fibroblasts Display Calcium-Dependent Potassium Currents
Author:
Publisher
Elsevier BV
Subject
Cell Biology
Reference17 articles.
1. Werner’s syndrome: An underdiagnosed disorder resembling premature aging
2. Werner Syndrome: A Molecular Genetic Hypothesis
3. A novel gene encoding a smooth muscle protein is overexpressed in senescent human fibroblasts
4. The gene responsible for Werner syndrome may be a cell division "counting" gene.
5. Suppression of calcium-dependent membrane currents in human fibroblasts by replicative senescence and forced expression of a gene sequence encoding a putative calcium-binding protein.
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1. Characterization of cellular senescence mechanisms in human corneal endothelial cells;Aging Cell;2011-12-29
2. The Progerias;Journal of Anti-Aging Medicine;2003-09
3. A lack of telomeric non-reciprocal recombination (TENOR) may account for the premature proliferation blockade of Werner's syndrome fibroblasts;Biogerontology;2003
4. A cell kinetic analysis of human umbilical vein endothelial cells;Mechanisms of Ageing and Development;2000-12
5. Fibroblasts cultured from venous ulcers display cellular characteristics of senescence;Journal of Vascular Surgery;1998-11
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