Chronic Nicotine Treatment Differentially Regulates Substance P and Tyrosine Hydroxylase Immunoreactivity in Substantia Nigra Ipsilateral to a Unilateral Lesion-Reference-Cited by-同舟云学术

Chronic Nicotine Treatment Differentially Regulates Substance P and Tyrosine Hydroxylase Immunoreactivity in Substantia Nigra Ipsilateral to a Unilateral Lesion

Published:1997-08 Issue:2 Volume:146 Page:575-586
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Janson Ann Marie,Fuxe Kjell

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference41 articles.

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2. Functional studies with substance P analogues: Effects of N-terminal, C-terminal, and C-terminus-extended analogues of substance P on nicotine-induced secretion and desensitization in cultured bovine adrenal chromaffin cells;Cheung;J. Neurochem.,1994

3. Nigrostriatal dopamine neurons are required to maintain basal levels of substance P in the rat substantia nigra;Cruz;Neuroscience,1989

4. Acute continuous exposure to cigarette smoke produces discrete changes in cholecystokinin and substance P levels in the hypothalamus and preoptic area of the male rat;Fuxe;Acta Physiol. Scand.,1985

5. Chronic nicotine treatment increases dopamine levels and reduces dopamine utilization in substantia nigra and in surviving forebrain dopamine nerve terminal systems after a partial di-mesencephalic hemitransection;Fuxe;Naunyn-Schmiedeberg's Arch. Pharmacol.,1990

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1. Nicotine induces tyrosine hydroxylase plasticity in the neurodegenerating striatum;Journal of Neurochemistry;2007-03-09

2. Neuroprotective strategies for basal ganglia degeneration: Parkinson's and Huntington's diseases;Progress in Neurobiology;2000-04