Repression of GAD Autoantigen Expression in Pancreas β-Cells by Delivery of Antisense Plasmid/PEG-g-PLL Complex-Reference-Cited by-同舟云学术

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Repression of GAD Autoantigen Expression in Pancreas β-Cells by Delivery of Antisense Plasmid/PEG-g-PLL Complex

Published:2001-10 Issue:4 Volume:4 Page:339-346
ISSN:1525-0016
Container-title:Molecular Therapy
language:en
Short-container-title:Molecular Therapy

Author:

Lee Minhyung,Han Sang-oh,Ko Kyung Soo,Koh Jae Joon,Park Jong-Sang,Yoon Ji-Won,Kim Sung Wan

Publisher

Elsevier BV

Subject

Drug Discovery,Pharmacology,Genetics,Molecular Biology,Molecular Medicine

Reference37 articles.

1. The pathogenesis of insulin-dependent diabetes mellitus;Atkinson;N. Engl. J. Med.,1994

2. Anti-glutamate decarboxylase and other antibodies at the onset of childhood IDDM: a population-based study;Verge;Diabetologia.,1994

3. Glutamate decarboxylase-, insulin-, and islet cell-anti-bodies and HLA typing to detect diabetes in a general population-based study of Swedish children;Hagopian;J. Clin. Invest.,1995

4. Autoantigens IA-2 and GAD in type I (insulin-dependent) diabetes;Leslie;Diabetologia.,1999

5. Cloning and primary structure of a human islet isoform of glutamic acid decarboxylase from chromosome 10;Karlsen;Proc. Natl. Acad. Sci. USA.,1991

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