Phosphorylation of ARC Is a Critical Element in the Antiapoptotic Effect of Anesthetic Preconditioning
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Anesthesiology and Pain Medicine
Reference32 articles.
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3. Sevoflurane preconditioning limits intracellularmitochondrial Ca2 in ischemic newborn myocardium.;Liu;Anesth Analg,2005
4. Isoflurane preconditioning protects against ischemia-reperfusion injury partly by attenuating cytochrome c release from subsarcolemmal mitochondria in isolated rat hearts.;Qian;Acta Pharmacol Sin,2005
5. ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases.;Koseki;Proc Natl Acad Sci USA,1998
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3. Role of apoptosis repressor with caspase recruitment domain (ARC) in cell death and cardiovascular disease;Apoptosis;2021-02
4. Sevoflurane Preconditioning Confers Delayed Cardioprotection by Upregulating AMP-Activated Protein Kinase Levels to Restore Autophagic Flux in Ischemia-Reperfusion Rat Hearts;Medical Science Monitor;2020-06-01
5. Loss of nuclear ARC contributes to the development of cardiac hypertrophy in rats;Acta Physiologica;2019-07-27
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