IC2 participates in the cooperative activation of outer arm dynein densely attached to microtubules
Author:
Affiliation:
1. Department of Life and Environmental Sciences, Faculty of Bioresource Sciences, Prefectural University of Hiroshima
2. Department of Biological Sciences, Chuo University
3. Department of Frontier Bioscience, Hosei University
Publisher
Japan Society for Cell Biology
Subject
Cell Biology,Molecular Biology,Physiology,General Medicine
Link
https://www.jstage.jst.go.jp/article/csf/48/2/48_23044/_pdf
Reference54 articles.
1. Afzelius, B.A. 2004. Cilia-related diseases. J. Pathol., 204: 470–477.
2. Al-Mutairi, D.A., Alsabah, B.H., Alkhaledi, B.A., Pennekamp, P., and Omran, H. 2022. Identification of a novel founder variant in DNAI2 cause primary ciliary dyskinesia in five consanguineous families derived from a single tribe descendant of Arabian Peninsula. Front. Genet., 13: 1017280.
3. Aoyama, S. and Kamiya, R. 2010. Strikingly fast microtubule sliding in bundles formed by Chlamydomonas axonemal dynein. Cytoskeleton (Hoboken), 67: 365–372.
4. Brokaw, C.J. and Kamiya, R. 1987. Bending patterns of Chlamydomonas flagella: IV. Mutants with defects in inner and outer dynein arms indicate differences in dynein arm function. Cell Motil. Cytoskeleton, 8: 68–75.
5. Dibella, L.M., Gorbatyuk, O., Sakato, M., Wakabayashi, K., Patel-King, R.S., Pazour, G.J., Witman, G.B., and King, S.M. 2005. Differential light chain assembly influences outer arm dynein motor function. Mol. Biol. Cell, 16: 5661–5674.
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