Specific association of TBK1 with the trans-Golgi network following STING stimulation
Author:
Affiliation:
1. Laboratory of Organelle Pathophysiology, Department of Integrative Life Sciences, Graduate School of Life Sciences, Tohoku University
Publisher
Japan Society for Cell Biology
Subject
Cell Biology,Molecular Biology,Physiology,General Medicine
Link
https://www.jstage.jst.go.jp/article/csf/47/1/47_21080/_pdf
Reference45 articles.
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2. Deng, Z., Chong, Z., Law, C.S., Mukai, K., Ho, F.O., Martinu, T., Backes, B.J., Eckalbar, W.L., Taguchi, T., and Shum, A.K. 2020. A defect in COPI-mediated transport of STING causes immune dysregulation in COPA syndrome. J. Exp. Med., 217: e20201045.
3. Dobbs, N., Burnaevskiy, N., Chen, D., Gonugunta, V.K., Alto, N.M., and Yan, N. 2015. STING Activation by Translocation from the ER Is Associated with Infection and Autoinflammatory Disease. Cell Host Microbe, 18: 157–168.
4. Duran, J.M., Campelo, F., van Galen, J., Sachsenheimer, T., Sot, J., Egorov, M.V., Rentero, C., Enrich, C., Polishchuk, R.S., Goni, F.M., Brugger, B., Wieland, F., and Malhotra, V. 2012. Sphingomyelin organization is required for vesicle biogenesis at the Golgi complex. EMBO J., 31: 4535–4546.
5. Fang, R., Jiang, Q., Guan, Y., Gao, P., Zhang, R., Zhao, Z., and Jiang, Z. 2021. Golgi apparatus-synthesized sulfated glycosaminoglycans mediate polymerization and activation of the cGAMP sensor STING. Immunity, 54: 962–975.e8.
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