Roles of MAPKKK ASK1 in Stress-Induced Cell Death.-Reference-Cited by-同舟云学术

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Roles of MAPKKK ASK1 in Stress-Induced Cell Death.

Published:2003 Issue:1 Volume:28 Page:23-29
ISSN:0386-7196
Container-title:Cell Structure and Function
language:en
Short-container-title:Cell Struct. Funct.

Author:

Takeda Kohsuke¹²³,Matsuzawa Atsushi²³,Nishitoh Hideki¹²³,Ichijo Hidenori¹²³

Affiliation:

1. Laboratory of Cell Signaling, Graduate School, Tokyo Medical and Dental University

2. Laboratory of Cell Signaling, Graduate School of Pharmaceutical Sciences, The University of Tokyo

3. Core Research for Evolutional Science and Technology, Japan Science and Technology Corporation

Publisher

Japan Society for Cell Biology

Subject

Cell Biology,Molecular Biology,Physiology,General Medicine

Link

http://www.jstage.jst.go.jp/article/csf/28/1/28_1_23/_pdf

Reference54 articles.

1. Integration of endoplasmic reticulum signaling in health and disease

2. Impairment of the Ubiquitin-Proteasome System by Protein Aggregation

3. Dynamic interaction of BiP and ER stress transducers in the unfolded-protein response

4. Proteasome Inhibition Leads to a Heat-shock Response, Induction of Endoplasmic Reticulum Chaperones, and Thermotolerance

5. Activation of Apoptosis Signal-Regulating Kinase 1 (ASK1) by the Adapter Protein Daxx

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