Involvement of Long Non-Coding RNA HOTAIR in the Regulation of ERα36-Mediated Epithelial Mesenchymal Transition in Tamoxifen-Resistant Human Breast Cancer-Reference-Cited by-同舟云学术

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Involvement of Long Non-Coding RNA HOTAIR in the Regulation of ERα36-Mediated Epithelial Mesenchymal Transition in Tamoxifen-Resistant Human Breast Cancer

Published:2024-03-31 Issue:1 Volume:3 Page:1-13
ISSN:2951-3359
Container-title:Drug Targets and Therapeutics
language:en
Short-container-title:DTT

Author:

Bui Quyen Thu¹,Im Ji Hye¹,Kim Suntae¹,Lim Sung Chul²,Kim Sang Kyum³,Kang Keon Wook¹^ORCID

Affiliation:

1. College of Pharmacy and Research Institute of Pharmaceutical Sciences, Seoul National University, Seoul, Korea

2. Department of Pathology, College of Medicine, Chosun University, Gwangju, Korea

3. College of Pharmacy, Chungnam National University, Daejeon, Korea

Funder

National Research Foundation of Korea

Publisher

Drug Targets and Therapeutics (DTT) - Pharmaceutical Society of Korea

Link

https://pdf.medrang.co.kr/DTT/2024/003/dtt-3-1-1.pdf

Reference45 articles.

1. Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo

2. LncRNA HOTAIR: A master regulator of chromatin dynamics and cancer

3. Essential role of Notch4/STAT3 signaling in epithelial–mesenchymal transition of tamoxifen-resistant human breast cancer

4. Reassessing Epithelial to Mesenchymal Transition as a Prerequisite for Carcinoma Invasion and Metastasis

5. Hormone resistance, invasiveness, and metastatic potential in breast cancer

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