Oncogene-dependent Tumor Suppression: Using the Dark Side of the Force for Cancer Therapy-Reference-Cited by-同舟云学术

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Oncogene-dependent Tumor Suppression: Using the Dark Side of the Force for Cancer Therapy

Published:2005-01-01 Issue:0 Volume:70 Page:263-273
ISSN:0091-7451
Container-title:Cold Spring Harbor Symposia on Quantitative Biology
language:en
Short-container-title:Cold Spring Harbor Symposia on Quantitative Biology

Author:

EVAN G.I.,CHRISTOPHOROU M.,LAWLOR E.A.,RINGSHAUSEN I.,PRESCOTT J.,DANSEN T.,FINCH A.,MARTINS C.,MURPHY D.

Publisher

Cold Spring Harbor Laboratory

Subject

Genetics,Molecular Biology,Biochemistry

Reference35 articles.

1. Activation of apoptosis associated with enforced myc expression in myeloid progenitor cells is dominant to the suppression of apoptosis by interleukin-3 or erythropoietin

2. Cancer and ageing: rival demons?

3. Temporal dissection of p53 function in vitro and in vivo

4. Expression analysis with oligonucleotide microarrays reveals that MYC regulates genes involved in growth, cell cycle, signaling, and adhesion

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1. Apoptotic cell death in disease—Current understanding of the NCCD 2023;Cell Death & Differentiation;2023-04-26

2. Role of ARK5 in cancer and other diseases (Review);Experimental and Therapeutic Medicine;2021-05-02

3. Calcium signalling links MYC to NUAK1;Oncogene;2017-11-06

4. BIM Is the Primary Mediator of MYC-Induced Apoptosis in Multiple Solid Tissues;Cell Reports;2014-09

5. FOXM1 (Forkhead box M1) in Tumorigenesis;Advances in Cancer Research;2013

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