Abstract
In this issue of Genes & Development, Abruzzi et al. (pp. 2374–2386) use chromatin immunoprecipitation (ChIP) tiling array assays (ChIP–chip) to show that physical interactions between circadian (≅24-h) clock machineries and genomes are more widespread than previously thought and provide novel insights into how clocks drive daily rhythms in global gene expression.
Publisher
Cold Spring Harbor Laboratory
Subject
Developmental Biology,Genetics
Reference40 articles.
1. Abruzzi KC , Rodriguez J , Menet JS , Desrochers J , Zadina A , Luo W , Tkachev S , Rosbash M . 2011. Drosophila CLOCK target gene characterization: Implications for circadian tissue-specific gene expression. Genes Dev (this issue). doi: 10.1101/gad.178079.111.
2. Stopping Time: The Genetics of Fly and Mouse Circadian Clocks
3. Regulating a Circadian Clock's Period, Phase and Amplitude by Phosphorylation: Insights from Drosophila
4. dCLOCK is present in limiting amounts and likely mediates daily interactions between the dCLOCK–CYC transcription factor and the PER–TIM complex;J Neurosci,2000
5. Baker CL , Loros JJ , Dunlap JC . 2011. The circadian clock of Neurospora crassa . FEMS Microbiol Rev. doi: 10.1111./j.1574-6976.2011.00288.x.
Cited by
9 articles.
订阅此论文施引文献
订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献