Homologous Recombination and Replication Fork Protection: BRCA2 and More!
Author:
Publisher
Cold Spring Harbor Laboratory
Subject
Genetics,Molecular Biology,Biochemistry
Reference92 articles.
1. ES cells do not activate p53-dependent stress responses and undergo p53-independent apoptosis in response to DNA damage
2. Average Risks of Breast and Ovarian Cancer Associated with BRCA1 or BRCA2 Mutations Detected in Case Series Unselected for Family History: A Combined Analysis of 22 Studies
3. BRCA2 acts as a RAD51 loader to facilitate telomere replication and capping
4. Replication stress: getting back on track
5. SMARCAL1 catalyzes fork regression and Holliday junction migration to maintain genome stability during DNA replication
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1. A novel role for the peptidyl-prolyl cis-trans isomerase Cyclophilin A in DNA-repair following replication fork stalling via the MRE11-RAD50-NBS1 complex;EMBO Reports;2024-06-28
2. DMC1 and RAD51 bind FxxA and FxPP motifs of BRCA2 via two separate interfaces;Nucleic Acids Research;2024-06-03
3. Improved detection of DNA replication fork-associated proteins;Cell Reports;2024-05
4. APLF facilitates interstrand DNA crosslink repair and replication fork protection to confer cisplatin resistance;Nucleic Acids Research;2024-03-23
5. CaMKK2 and CHK1 phosphorylate human STN1 in response to replication stress to protect stalled forks from aberrant resection;Nature Communications;2023-11-30
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