Ideal efficacy photoswitches for TRPC4/5 channels harness high potency for spatiotemporally-resolved control of TRPC function in live tissues

Author:

Müller Markus,Niemeyer Konstantin,Ojha Navin K.ORCID,Porav Sebastian A.ORCID,Vinayagam DeivanayagabarathyORCID,Urban NicoleORCID,Büchau Fanny,Oleinikov Katharina,Makke Mazen,Bauer Claudia C.ORCID,Johnson Aidan V.ORCID,Muench Stephen P.ORCID,Zufall FrankORCID,Bruns Dieter,Schwarz Yvonne,Raunser StefanORCID,Leinders-Zufall TreseORCID,Bon Robin S.ORCID,Schaefer MichaelORCID,Thorn-Seshold OliverORCID

Abstract

ABSTRACTDirectly probing the endogenous biological roles of target proteins with high spatial and temporal resolution, as non-invasively and reproducibly as possible, is a shared conceptual goal for research across many fields, as well as for targeted therapies. Here we describe the rational conceptual design and test-case practical implementation of a photopharmacological paradigm to empower high-performance photomodulation studiesin vivo. TRPC4/5 ion channels are involved in many spatiotemporally resolved circuits, from pain and anxiety, to reproductive signaling, digestion, and obesity. To unpick their biology requires spatiotemporally precise tools, which were lacking. We developed “ideal efficacy photoswitch” ligands to control their diverse functionsin situ. TheseEZ-photoswitchable ligands bias TRPC[4]/5 channel activity with exquisite photocontrol, from strong agonism under 360 nm, to low agonism at 385 nm, to strong antagonism at 410-460 nm. Cryo-EM structures of both TRPC4 and TRPC5 with bothZ-agonists andE-antagonists support the rationale for efficacy switching through competitiveE/Zisomer binding. Crucially, since theE/Zratio is exclusively determined by the light wavelength applied,their channel photocontrol is exclusively wavelength-dependent, yet drug-concentration-independent: so is reproducible from cell culture to >millimetre-depth tissues. Indeed, we were able to photocontrol both direct and downstream TRPC4/5 biology in cell lines or primary cells in culture, from calcium flux, to primary neuron excitability and adrenaline release; and even in tissues, photoswitching small intestine motility and peristalsis. The TRPC4/5 ligands we develop will thus unlock a range of high-precision investigations in TRP biology. More broadly, we propose that the success of this efficacy photoswitch program, from concept to tissue level translation, is mainly a consequence of how biology has evolved proteins for efficacy control. We therefore foresee that a variety of functionally responsive protein targets, not only sensory and signaling ion channels and receptors, will be amenable to similarly high-performance photocontrol evenin vivo, if a new generation of reagent development adopts this paradigm ofideal efficacy photoswitching.Table of Contents Graphic

Publisher

Cold Spring Harbor Laboratory

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