The effect of circulating neutralizing antibodies on the replication of SARS-CoV-2 variants following post-vaccination infections

Author:

Garcia-Knight Miguel A.ORCID,Kelly J. Daniel,Lu Scott,Tassetto Michel,Goldberg Sarah A.,Zhang Amethyst,Pineda-Ramirez Jesus,Anglin Khamal,Davidson Michelle C.,Chen Jessica Y.,Fortes-Cobby Maya,Park Sara,Martinez Ana,So Matthew,Donovan Aidan,Viswanathan Badri,Richardson Eugene T.ORCID,McIlwain David R.,Gaudilliere Brice,Rutishauser Rachel L.,Chenna Ahmed,Petropoulos Christos,Wrin Terri,Deeks Steve G.ORCID,Abedi Glen R.,Saydah Sharon,Martin Jeffrey N.ORCID,Briggs-Hagen Melissa,Midgley Claire M.,Peluso Michael J.,Andino Raul

Abstract

ABSTRACTThe impact of pre-existing neutralizing antibodies (NAbs) titers on SARS-CoV-2 viral shedding dynamics in post-vaccination infection (PVI) are not well understood. We characterized viral shedding longitudinally in nasal specimens in relation to baseline (pre/peri-infection) serum neutralizing antibody titers in 125 participants infected with distinct SARS-CoV-2 variants. Among 68 participants who had received vaccinations, we quantified the effect of baseline serum NAb titers on maximum viral RNA titers and on the duration of infectivity. Baseline NAb titers were higher and efficiently targeted a broader range of variants in participants who received one or two monovalent ancestral booster vaccinations compared to those with a full primary vaccine series. In participants with Delta variant infections, baseline NAb titers targeting Delta were negatively correlated with maximum viral RNA copies. Per log10increase in baseline NAb IC50, maximum viral load was reduced -2.43 (95% confidence interval [CI] -3.76, -1.11) log10 N copies and days of infectious viral shedding were reduced -2.79 [95% CI: -4.99, -0.60] days. By contrast, in those with Omicron infections (BA.1, BA.2, BA.4 or BA.5 lineages) baseline NAb responses against Omicron lineages did not predict viral outcomes. Our results provide robust estimates of the effect of baseline NAbs on the magnitude and duration of nasal viral replication after PVI (albeit with an unclear effect on transmission) and show how immune escape variants efficiently evade these modulating effects.

Publisher

Cold Spring Harbor Laboratory

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