Multitasking Kinase RIPK1 Regulates Cell Death and Inflammation
Author:
Publisher
Cold Spring Harbor Laboratory
Subject
General Biochemistry, Genetics and Molecular Biology
Reference203 articles.
1. The Pseudokinase MLKL and the Kinase RIPK3 Have Distinct Roles in Autoimmune Disease Caused by Loss of Death-Receptor-Induced Apoptosis
2. RIPK1 prevents TRADD-driven, but TNFR1 independent, apoptosis during development
3. Ubiquitin-Mediated Regulation of RIPK1 Kinase Activity Independent of IKK and MK2
4. Identification of TBK1 complexes required for the phosphorylation of IRF3 and the production of interferon β
5. A FADD-dependent innate immune mechanism in mammalian cells
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1. Crosstalk and Prospects of TBK1 in Inflammation;Immunological Investigations;2024-08-28
2. Blockade of IKK signaling induces RIPK1-independent apoptosis in human macrophages;PLOS Pathogens;2024-08-26
3. TBK1 and IKKε protect target cells from IFNγ-mediated T cell killing via an inflammatory apoptotic mechanism;2024-08-08
4. The RIPK1 death domain restrains ZBP1- and TRIF-mediated cell death and inflammation;Immunity;2024-07
5. RIPK1: Inflamed if you do, inflamed if you don’t;Immunity;2024-07
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