ER-Phagy: Quality and Quantity Control of the Endoplasmic Reticulum by Autophagy
Author:
Publisher
Cold Spring Harbor Laboratory
Subject
General Biochemistry, Genetics and Molecular Biology
Reference136 articles.
1. TEX264 Is an Endoplasmic Reticulum-Resident ATG8-Interacting Protein Critical for ER Remodeling during Nutrient Stress
2. Basal autophagy maintains pancreatic acinar cell homeostasis and protein synthesis and prevents ER stress
3. The Protease Ste24 Clears Clogged Translocons
4. Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum
5. Autophagy Counterbalances Endoplasmic Reticulum Expansion during the Unfolded Protein Response
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