Airborne Fine Particulate Matter Induces Oxidative Stress and Inflammation in Human Nasal Epithelial Cells
Author:
Affiliation:
1. Department of Otolaryngology, Huadong Hospital, Fudan University
2. Center for Atmospheric Chemistry Study, Department of Environmental Science and Engineering, Fudan University
Publisher
Tohoku University Medical Press
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/tjem/239/2/239_117/_pdf
Reference27 articles.
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2. Corren, J. (2013) Role of interleukin-13 in asthma. Curr. Allergy Asthma Rep., 13, 415-420.
3. Delfino, R.J., Sioutas, C. & Malik, S. (2005) Potential role of ultrafine particles in associations between airborne particle mass and cardiovascular health. Environ. Health Perspect., 113, 934-946.
4. Deng, X., Zhang, F., Rui, W., Long, F., Wang, L., Feng, Z., Chen, D. & Ding, W. (2013) PM2.5-induced oxidative stress triggers autophagy in human lung epithelial A549 cells. Toxicol. In Vitro, 27, 1762-1770.
5. Ghio, A.J. & Devlin, R.B. (2001) Inflammatory lung injury after bronchial instillation of air pollution particles. Am. J. Respir. Crit. Care Med., 164, 704-708.
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