Reduction of mutant ataxin-7 expression restores motor function and prevents cerebellar synaptic reorganization in a conditional mouse model of SCA7-Reference-Cited by-同舟云学术

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Reduction of mutant ataxin-7 expression restores motor function and prevents cerebellar synaptic reorganization in a conditional mouse model of SCA7

Published:2012-11-29 Issue:5 Volume:22 Page:890-903
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Furrer S. A.,Waldherr S. M.,Mohanachandran M. S.,Baughn T. D.,Nguyen K.-T.,Sopher B. L.,Damian V. A.,Garden G. A.,La Spada A. R.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/22/5/890/1722759/dds495.pdf

Reference22 articles.

1. Molecular pathogenesis and cellular pathology of spinocerebellar ataxia type 7 neurodegeneration

2. Trinucleotide Repeat Disorders

3. Reversal of Neuropathology and Motor Dysfunction in a Conditional Model of Huntington's Disease

4. Recovery from Polyglutamine-Induced Neurodegeneration in Conditional SCA1 Transgenic Mice

5. Spinocerebellar Ataxia Type 7 Cerebellar Disease Requires the Coordinated Action of Mutant Ataxin-7 in Neurons and Glia, and Displays Non-Cell-Autonomous Bergmann Glia Degeneration

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2. Experimental Neurotransplantation for Cerebellar Ataxias;Contemporary Clinical Neuroscience;2023

3. The cannabinoid antagonist, AM251 attenuates ataxia related deficiencies in a cerebellar ataxic model;International Journal of Neuroscience;2022-09-30

4. The Molecular Basis of Spinocerebellar Ataxia Type 7;Frontiers in Neuroscience;2022-03-24

5. A Subpopulation of Microglia Generated in the Adult Mouse Brain Originates from Prominin-1-Expressing Progenitors;The Journal of Neuroscience;2021-08-11

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