Bloodstream infection by two subpopulations of Klebsiella pneumoniae ST1685 carrying KPC-33 or KPC-14 following ceftazidime/avibactam treatment: considerations regarding acquired heteroresistance and choice of carbapenemase detection assay
Author:
Affiliation:
1. Microbiology and Virology Unit, University Hospital Città della Salute e della Scienza di Torino, Turin, Italy
Funder
internal funding
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)
Link
http://academic.oup.com/jac/article-pdf/75/10/3075/33885400/dkaa283.pdf
Reference10 articles.
1. Resistance to ceftazidime-avibactam and underlying mechanisms;Wang;J Glob Antimicrob Resist,2019
2. Emergence of ceftazidime-avibactam resistance due to plasmid-borne blaKPC-3 mutations during treatment of carbapenem-resistant Klebsiella pneumoniae infections;Shields;Antimicrob Agents Chemother,2017
3. Mutations in blaKPC-3 that confer ceftazidime-avibactam resistance encode novel KPC-3 variants that function as extended-spectrum β-lactamases;Haidar;Antimicrob Agents Chemother,2017
4. Genomic characterization of a KPC-23-producing Klebsiella pneumoniae ST258 clinical isolate resistant to ceftazidime-avibactam;Galani;Clin Microbiol Infect,2019
5. In vivo evolution of resistant subpopulations of KPC-producing Klebsiella pneumoniae during ceftazidime/avibactam treatment;Gaibani;J Antimicrob Chemother,2018
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